![]() In February of this year, researchers published an article outlining how a drug derived from the spiny dogfish could completely suppress the toxic effect of the Parkinson’s associated protein Alpha Synuclein ( Click here to read that post).Īnd then in May (JUST 3 MONTHS LATER!!!), a biotech company called Enterin Inc. Just as I manage to digest something new from one area of research, two or three other publications pop up in different areas.īut it is the shear speed with which things are moving now in the field of Parkinson’s research that is really mind boggling! Not just here on the blog, but also with regards to the ever increasing number of research articles in the “need to read” pile on my desk. So much is happening so quickly that it is quite simply difficult to keep up. I have said several times in the past that the pace of Parkinson’s disease research at the moment is overwhelming. Technological progress – looking inside the brain. In today’s post we will review and discuss the results, and look at what happens next. Suddenly the distant future is feeling not so ‘distant’. So imagine my surprise when an Italian research group last weekend published a new research report in which they used this futurist technology to correct a mouse model of Parkinson’s disease. At the time I considered this technology way off at some point in the distant future. In July of this year, I wrote about a new class of engineered viruses that could potentially allow us to treat conditions like Parkinson’s disease using a non-invasive, gene therapy approach ( Click here to read that post). In addition to looking at current Parkinson’s disease research on this website, I like to look at where technological advances are taking us with regards to future therapies. Recently, some Canadian scientists discovered something interesing about the immune system and it relates to Parkinson’s.Ĭontinue reading “Something LRRKing in the immune system” → AAV-PHP.B: The future is apparently now Luckily, our immune systems are pretty robust – doing battle on a moment-to-moment basis with all manner of pathogenic agents. After a summer of salads and light food, there is nothing better that entering a warm cottage or pub, and smelling the hearty food (my wife if French – we navigate based on the quality of eateries).īut there is a down side to autumn: The start of the flu season. #Wordfast pro 4 msi license#I love the long, slow afternoon strolls and anticipation of the festive season to come.īut most of all I love the license to eat all the good wintery food. In today’s post, we will discuss what LRRK2 is, review the new research, and explore what the sex difference could mean in terms of Parkinson’s. And the researchers found that this effect was most prominent in female mice in particular.Īnd curiously, when the mice are infected with a dangerous virus, female mice with the Parkinson’s-associated LRRK2 mutation fared worse than their male counterparts. Canadian scientists recently reported that mice with a specific genetic variation – in the Parkinson’s-associated LRRK2 gene – differ in how they are able to deal with bacterial and viral infections.Ĭuriously, mice with the Parkinson’s-associated LRRK2 mutation could handle a bacterial infection better than normal mice, while mice with no LRRK2 protein struggled against the infection. ![]()
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